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Under pathological conditions, they may initially exert neuroprotective effects, yet prolonged activation often results in metabolic disturbances and neurotoxic gliotransmission, highlighting their dual role in CNS pathology and their potential in therapy [293]

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This predicts that the K D approach a plateau close to the value recorded for a single histidine residue when the linker is very long - and by extrapolation the ROS production would decrease similarly